永久不封国产毛片_亚洲欧美人成综合在线另类_国产 中文 制服丝袜 另类_久欠精品国国产99国产精20_久久国产乱子伦精品免费不卡_日韩中文字幕中文无码_孕妇奶水仑乱A级毛片免费看_四虎成人免费精品影库视频 _久久国产精品免费高清_91在线播放一区二区_日本XXXXX片免费观看喷水_国产名模A∨精品视频_1024你懂得金沙久久一区_亚洲国产精品Va在线观看牛牛_大香伊久久国产

歡迎來(lái)到北京博奧森生物技術(shù)有限公司網(wǎng)站!
咨詢熱線

4009019800

當(dāng)前位置:首頁(yè)  >  新聞資訊  >  【2024年2月文獻(xiàn)戰(zhàn)報(bào)】Bioss抗體新增高分文獻(xiàn)精彩呈現(xiàn)

【2024年2月文獻(xiàn)戰(zhàn)報(bào)】Bioss抗體新增高分文獻(xiàn)精彩呈現(xiàn)

更新時(shí)間:2024-05-28  |  點(diǎn)擊率:365

截止目前,引用Bioss產(chǎn)品發(fā)表的文獻(xiàn)共29281篇,總影響因子141751.15分,發(fā)表在Nature, Science, Cell以及Immunity等頂級(jí)期刊的文獻(xiàn)共68篇,合作單位覆蓋了清華、北大、復(fù)旦、華盛頓大學(xué)、麻省理工學(xué)院、東京大學(xué)以及紐約大學(xué)等國(guó)際研究機(jī)構(gòu)上百所。

我們每月收集引用Bioss產(chǎn)品發(fā)表的文獻(xiàn)。若您在當(dāng)月已發(fā)表SCI文章,但未被我公司收集,請(qǐng)致電Bioss,我們將贈(zèng)予現(xiàn)金鼓勵(lì),金額標(biāo)準(zhǔn)請(qǐng)參考“發(fā)文章 領(lǐng)獎(jiǎng)金"活動(dòng)頁(yè)面。

近期收錄2024年2月引用Bioss產(chǎn)品發(fā)表的文獻(xiàn)共363篇(圖一,綠色柱),文章影響因子(IF) 總和高達(dá)2298.3,其中,10分以上文獻(xiàn)45篇(圖二)。

【2024年2月文獻(xiàn)戰(zhàn)報(bào)】Bioss抗體新增高分文獻(xiàn)精彩呈現(xiàn)

圖一


【2024年2月文獻(xiàn)戰(zhàn)報(bào)】Bioss抗體新增高分文獻(xiàn)精彩呈現(xiàn)

圖二



本文主要分享引用Bioss產(chǎn)品發(fā)表文章至NatureImmunityCancer Cell等期刊的5篇 IF>15 的文獻(xiàn)摘要,讓我們一起欣賞吧。




Molecular Cancer [IF=37.3]

【2024年2月文獻(xiàn)戰(zhàn)報(bào)】Bioss抗體新增高分文獻(xiàn)精彩呈現(xiàn)

文獻(xiàn)引用產(chǎn)品:bsm-33070M

Ki-67 Mouse mA | IHC

作者單位:重慶醫(yī)科大學(xué)

摘要:CircPDHK1 was upregulated in ccRCC tissues and closely related to WHO/ISUP stage, T stage, distant metastasis, VHL mutation and Ki-67 levels. CircPDHK1 had a functional internal ribosome entry site (IRES) and encoded a novel peptide PDHK1-241aa. Functionally, we confirmed that PDHK1-241aa and not the circPDHK1 promoted the proliferation, migration and invasion of ccRCC. Mechanistically, circPDHK1 was activated by HIF-2A at the transcriptional level. PDHK1-241aa was upregulated and interacted with PPP1CA, causing the relocation of PPP1CA to the nucleus. This thereby inhibited AKT dephosphorylation and activated the AKT-mTOR signaling pathway.



Cellular & Molecular Immunology [IF=24.1]

【2024年2月文獻(xiàn)戰(zhàn)報(bào)】Bioss抗體新增高分文獻(xiàn)精彩呈現(xiàn)

文獻(xiàn)引用抗體:

bs-2789R; Tap1 Rabbit pAb | FC

bs-2374R; TAP2 Rabbit pAb | FC

作者單位:北京大學(xué)

要:CD4+ T cells can "help" or "license" conventional type 1 dendritic cells (cDC1s) to induce CD8+ cytotoxic T lymphocyte (CTL) anticancer responses, as proven in mouse models. We recently identified cDC1s with a transcriptomic imprint of CD4+ T-cell help, specifically in T-cell-infiltrated human cancers, and these cells were associated with a good prognosis and response to PD-1-targeting immunotherapy. Here, we delineate the mechanism of cDC1 licensing by CD4+ T cells in humans. Activated CD4+ T cells produce IFNβ via the STING pathway, which promotes MHC-I antigen (cross-)presentation by cDC1s and thereby improves their ability to induce CTL anticancer responses. In cooperation with CD40 ligand (L), IFNβ also optimizes the costimulatory and other functions of cDC1s required for CTL response induction. IFN-I-producing CD4+ T cells are present in diverse T-cell-infiltrated cancers and likely deliver “help" signals to CTLs locally, according to their transcriptomic profile and colocalization with “helped/licensed" cDCs and tumor-reactive CD8+ T cells. In agreement with this scenario, the presence of IFN-I-producing CD4+ T cells in the TME is associated with overall survival and the response to PD-1 checkpoint blockade in cancer patients.



ADVANCED FUNCTIONAL MATERIALS [IF=19.0]

【2024年2月文獻(xiàn)戰(zhàn)報(bào)】Bioss抗體新增高分文獻(xiàn)精彩呈現(xiàn)

文獻(xiàn)引用產(chǎn)品:bs-4917R

Osteocalcin Rabbit pAb | IF

作者單位:北京大學(xué)口腔醫(yī)院

摘要:Utilization of electro-responsive biomaterials with antibacterial properties is advantageous for facilitating septic wound healing and tissue regeneration. However, the dose-response effects of electrical stimuli from these materials against bacteria are not rigorously characterized, and achieving synergy of bactericidal and pro-regenerative effects of biomaterials remains a major challenge. Here, a graded series of flexible BaTiO3/P(VDF-TrFE) electroactive nanocomposite membranes (EMs) are developed with varying surface charge intensities, to serve as antibacterial dressing for septic wound healing. EMs display broad-spectrum antibacterial effects against both Gram-positive and Gram-negative bacteria in a dose-dependent manner, depending on the magnitude of their surface electrical potential. Mechanistically, the surface charge of EMs increase intracellular levels of reactive oxygen species within bacteria cells, which in turn caused oxidative damage to the bacterial membrane, thereby suppressing bacterial activity and biofilm formation. Moreover, in vivo studies demonstrated that EMs effectively inhibited S. aureus infection and accelerated wound healing in a mouse skin defect model, as well as ameliorated P. gingivalis-mediated periodontal inflammation in a mouse periodontitis model. Hence, this study optimizes the antibacterial properties of electroactive materials and characterizes the dose-response effects of surface electrical charge against bacteria, thus validating the therapeutic applications of electroactive biomaterials in combating bacterial infection.


ACS Nano [IF=17.1]

【2024年2月文獻(xiàn)戰(zhàn)報(bào)】Bioss抗體新增高分文獻(xiàn)精彩呈現(xiàn)

文獻(xiàn)引用抗體:

bs-0292P; BSA-V

D-9106; DAPI

bsm-33070M; Ki-67 Mouse mAb | IF

作者單位:北京大學(xué)

摘要:Cancer progression and treatment-associated cellular stress impairs therapeutic outcome by inducing resistance. Endoplasmic reticulum (ER) stress is responsible for core events. Aberrant activation of stress sensors and their downstream components to disrupt homeostasis have emerged as vital regulators of tumor progression as well as response to cancer therapy. Here, an orchestrated nanophotoinducer (ERsNP) results in specific tumor ER-homing, induces hyperthermia and mounting oxidative stress associated reactive oxygen species (ROS), and provokes intense and lethal ER stress upon near-infrared laser irradiation. The strengthened “dying" of ER stress and ROS subsequently induce apoptosis for both primary and abscopal B16F10 and GL261 tumors, and promote damage-associated molecular patterns to evoke stress-dependent immunogenic cell death effects and release “self-antigens". Thus, there is a cascade to activate maturation of dendritic cells, reprogram myeloid-derived suppressor cells to manipulate immunosuppression, and recruit cytotoxic T lymphocytes and effective antitumor response. The long-term protection against tumor recurrence is realized through cascaded combinatorial preoperative and postoperative photoimmunotherapy including the chemokine (C–C motif) receptor 2 antagonist, ERsNP upon laser irradiation, and an immune checkpoint inhibitor. The results highlight great promise of the orchestrated nanophotoinducer to exert potent immunogenic cell stress and death by reinforcing ER stress and oxidative stress to boost cancer photoimmunotherapy.


Advanced Science [IF=15.1]

【2024年2月文獻(xiàn)戰(zhàn)報(bào)】Bioss抗體新增高分文獻(xiàn)精彩呈現(xiàn)

文獻(xiàn)引用抗體:

bs-0292P-FITC; BSA / FITC | IF

bsm-60235R; Keratin 6 Recombinant Rabbit mAb | IF

作者單位:南方醫(yī)科大學(xué)

摘要:To address current challenges in effectively treating large skin defects caused by trauma in clinical medicine, the fabrication, and evaluation of a novel radially aligned nanofiber scaffold (RAS) with dual growth factor gradients is presented. These aligned nanofibers and the scaffold's spatial design provide many all-around “highways" for cell migration from the edge of the wound to the center area. Besides, the chemotaxis induced by two growth factor gradients further promotes cell migration. Incorporating epidermal growth factor (EGF) aids in the proliferation and differentiation of basal layer cells in the epidermis, augmenting the scaffold's ability to promote epidermal regeneration. Concurrently, the scaffold-bound vascular endothelial growth factor (VEGF) recruits vascular endothelial cells at the wound's center, resulting in angiogenesis and improving blood supply and nutrient delivery, which is critical for granulation tissue regeneration. The RAS+EGF+VEGF group demonstrates superior performance in wound immune regulation, wound closure, hair follicle regeneration, and ECM deposition and remodeling compared to other groups. This study highlights the promising potential of hierarchically assembled nanofiber scaffolds with dual growth factor gradients for wound repair and tissue regeneration applications.





在线综合 亚洲 欧美中文字幕| 国产尤物AV尤物在线观看不卡| 日韩视频精品在线观看| 综合影视国产无码| 人人人干干人人干| 温婉少妇玩3p| 激情小说图片亚洲首页| 精品亚洲黄色片 国产精品导航一区二区 | 丁香五月性| 18啪啪手机免费性爱| 99久久久久| 亚洲中文一区二区三区| 婷婷成人五月天| 东亚亚洲无码高清| 91久久免费视频互動交流| 精品一区二区亚洲国产| 精品中文字幕第一页| 69精品人人人人| 国产浮力影院第1页| 在线观看亚洲成人精品| 97福利视频| 国语av最新自产拍在线观看| 夜夜操美女| 国产福利精品98视频| 国产情侣自拍在线播放| 久久一区二区高清免费| 国产精品白丝在线播放| 婷婷五月天成人网| 欧美麻豆成人同性GⅤ在线| 一区二区三区高清天码| 欧美视频一| 97人人草| 99啪啪| 香蕉久久国产AV一区二区| 欧美激情中文字幕另类小说| 日本十八禁免费看污网站| 亚洲av成人精品一区| 99啪啪| 成人短视频在线观看| 亚洲国产无码精品首页久久久| 国产区91柔拿会所技师| 日本不卡高清视频| www.大香| 97摸视频| 色婷五月| 色丁香久久| 婷婷五月天AV| 张柏芝国产一区在线观看| 呦呦一区| 一本色道久久天天射天天干| 老司机深夜18禁污污网站| 蜜乳成人AV| 成人无码电影在线观看网| 中国少妇XXXX做受| 黄片色区软件| 性爱av在线免费观看| 97人人操人人干| 亚洲av噜噜噜噜噜噜| 性无码专区2020| 欧美日韩另类在线| 亚洲成熟国产精品美女| 国产一区二区啪啪视频| 久久五月婷| 日韩激情无码影院| 日本成人A片免费看| 熟女色综合久久| 五月天丁香网| 无码一区二区精品视频久久久春药 | av日韩手机在线影视| 偷窥自拍A片| 大学生美女口爆| 免费一级精品啪啪视频| 国产麻豆一级精品视频| 大学生美女口爆| 免费公开人人操| 久久9视频| 日韩激情毛片一级久久久| 亚洲αv一区二区三区| 色99视频| 久久91视频| 亚洲人妻av| 天天色天天干天天射| 91色久| 天堂俺去俺来也www久久婷婷| 久草福利在线资源站| 老司机午夜精品视频| 久草免费福利在线播放| 激情开心五月天| 日本欧美一区二区三区免费| 色婷婷丁香五月| 八人操人人摸人人看| 亚欧色图在线激情| 亚洲 国产 精品一区| 国产无码三级视频在线观看| 一级久久性爱视频| 久久欲| 五月婷视频| 入口操逼网站| av强奸乱轮| A级在线视频| 国产福利精品最新在线| 黄片国产精品一区二区| 天天激清| 在线播放中文字幕| Blackedraw视频一区二区| 99久久精品欧美国产| 亚洲经典啪啪| 蜜臀操逼黄色视频操的好爽| 天天色天天干天天爱| 欧美激情激情xxxx欧美专区| 日本欧美国内在线| 欧美视频边做饭边橾| 一线黄色免费性爱片| 欧美精品久久久久久久丰满| 99热免费| 天天做天天爱| 99热91| 亚洲九区| 伊人久久婷婷| 久久ww| 99热色精品| 99在线精品观看99| 六月婷婷色综合| 丁香五月天堂| 天天干,天天日| 大香蕉啪啪啪| 精品人妻中文字幕高清| 日韩在线观看三级电影| 操国产高清| 91色色网站| 操b在线观看| 日韩在线欧美精品一区二区| 成人无码在线视频网站| 日本三级A片网站com| av强奸乱轮| 插日本熟女视频| 欧美日韩性爱视屏免费看了| 国产热RE99久久6国产精品首| 日本中文字幕在线视频 | 把腿张开老子CAO烂你| 综合操逼| 国内毛片免费h片在线| 97人人模人人爽人人| 婷婷丁香六月| 91丨国产丨白浆秘 洗澡动漫| 国产精品福利资源在线尤物| 亚洲密乳AV| 国产三区免费在线观看| 天美麻花大全视频| 亚洲高清视频在线免费观看| 日韩无码精品综合久久| 免费在线黄片视频| 色小视频蜜乳| 国产无马在线| 久久久免费的精品| 狠狠综合网| 在线观看免费视频国产| 日韩 欧美 另类 人妻| 亚洲av无码成人精品国产| 五月天精品| 免费一级精品啪啪视频| 亚欧高清v| 中国操逼无码| 国产亚洲禁久一区二区| 国产高清亚洲日韩一区| 国产亚洲女v在线观看| 精品一区二区成人动漫| 蜜乳视频网站| 国产亚洲精品自在线亚洲情侣| 欧美v亚洲v综合v国产v妖精| 一区二区三区免费岛国片| 丁香六月婷| 久久精品中文字幕无码l| 熟妇乱伦一区二区| 九色在线熟女国产黑人| 国产精品ww久久| 人人操人人操草草| 超碰精品日韩欧美国产| 大伊香蕉在线视频免费| 欧美性爱日韩高清| 少妇滛荡视频| 国产精品自拍xxxx| 国产1024在线播放| 精品国产久热在线观看| 自拍偷拍亚洲熟女妇人精品| 五月激情天| 国产欧美亚洲精品a第2页| 99老司机精品视频在线观看| 五月婷婷激情网| 日本精品第一视频在'| 久久直播国产| 无码区蜜乳| 又粗又长又大国产不卡| 欧美一级A片在线看视频性色| 日本熟女免费視颖| 狠狠操,使劲操| 日韩兔费看黄片| 色婷婷久久| 国产精品老师| 国产福利精品最新在线 | 色色五月天婷婷| 欧美顶级黄片AAAAA在线免费看| 白天啪啪晚上啪啪视频| 天天激情综合站| www.亚洲成人一区| 日韩欧美性吧婷婷乱伦大香蕉| 成人综合久久精品色婷婷| 精品一啪| 国产亚洲精品第一最新| 久久久无码av精| 在线中文字幕极品av| 国产精品自在线发布| 久久综合资源一区二区| xxxx网站亚洲精品| 香蕉久久AⅤ...| 乱伦图av| 唐山老熟妇露脸啪啪叫| 精品伊人久久久大香线蕉小说| 亚洲αv一区二区三区| α√在线| 天天精品| 九九久久综合| 91福利网在线观看| 国产精品盗摄 偷窥盗摄| 亚洲好看强奸乱伦| 精品人体无圣光凹凸| 一区二区三区免费岛国片| 国产亚州高清国产拍精| 女人被添高潮免费视频| 丁香五月激情综合| 26uuu最新| 午夜激情成人在线观看| 久久亚洲AV成人精品无码| 91丨熟女丨丰满熟女| 国内毛片无码一级毛片| 秋霞Av理论一级在线| 日韩无码操逼片| 天堂中文日本在线观看| 91丨九色丨国产打屁股| 欧美v亚洲v综合v国产v妖精| 精品欧美А∨无码黑人大荫蒂| 成人5码视频| 另类视频在线| www.yeyecao| 92人人操人人| av毛片aaaaa免费看| 久操网址| 97在线精品| 亚洲第一免费视频| 欧美做爰无码A片视频| 三级日韩一区二区三区| 国产亚洲精品美女久久久久久2021| 日韩少妇一区二区三区| 成人一二| 国产1769在线| 91成人高清在线观看| 手机看av网站在线看| 日本孕妇一区二区视频操逼免费看 | 激情久久久| 中文字幕AV乱伦| 99综合| 亚洲精品亚洲人成在线麻豆| 日本操逼视频免费| A片 AV一级在线播放观看免费 | 亚洲国产成人精品999| 天堂8在线新版官网| 肏逼福利网站| 91蜜桃婷婷狠狠久久综合9色| 插穴性爱视频在线观看| 国产日韩精品suv| 丁香九月激情| 国产69精品久久久久99尤物| 色吧5亚洲| 婷婷综合激情| 欧美成人黄网色网站| 少妇人妻好深太紧了vr91| 久久AV无码AV| 九九RE视频在线精品| 亚洲成人一区二区精品| 麻豆国产视频精品观看| 伊人国产成人av网站| 日本3级一区二区免费| 国产精品一区二区三区,亚洲综合| 伊人一级免费黄片| 五月婷婷大香蕉| www.色婷婷色综合| aⅴ日韩成人电影av在线免费看av大全| 色在线综合| 影音先锋每日最新资源在线观看| 亚洲综合另类| 操操碰| 亚洲成人免费在线| 色五月丁香五月| 日本道久久综合色色| 日韩欧美俄罗斯A片| 日韩欧美一级特黄大片| 欧美另类精品xxxx| 天天综合网日韩7799| 一起草日韩| 国产AV无码AV| 手机在线看片免费人成视频| 丰满高潮18xxxx| 国产精品岛国片在线观看| 天天操天天舔| www.超碰在线| 久久思思热| 成人av性爱电影在线观看| 青娱乐老司机视频| 成人乱人伦一区二区| 亚洲成人免费中文字幕| 久久精品国产亚洲AV片多多| 亚洲性爱无码乱伦av| 欧美日韩第一页| 99久久久久| 亚洲麻豆av一区二区| 亚洲精品白浆高清久久久久久 | 久久草草亚洲蜜桃臀| 中文人妻av高清一区| 亚洲免费成人精品电影| 动漫爆乳3D奶水一区在线观看 | 国产精品成人无码av无码免费| 一级性爱啪啪视频| 先锋激情∨在线视频播放| 国产亚洲美日韩Aⅴ中文字幕无码成人| 超碰成人公开| 久操不卡视频| 91无码西班牙视频在线| 亚洲无码成人精品|