永久不封国产毛片_亚洲欧美人成综合在线另类_国产 中文 制服丝袜 另类_久欠精品国国产99国产精20_久久国产乱子伦精品免费不卡_日韩中文字幕中文无码_孕妇奶水仑乱A级毛片免费看_四虎成人免费精品影库视频 _久久国产精品免费高清_91在线播放一区二区_日本XXXXX片免费观看喷水_国产名模A∨精品视频_1024你懂得金沙久久一区_亚洲国产精品Va在线观看牛牛_大香伊久久国产

歡迎來(lái)到北京博奧森生物技術(shù)有限公司網(wǎng)站!
咨詢熱線

4009019800

當(dāng)前位置:首頁(yè)  >  新聞資訊  >  【2024年2月文獻(xiàn)戰(zhàn)報(bào)】Bioss抗體新增高分文獻(xiàn)精彩呈現(xiàn)

【2024年2月文獻(xiàn)戰(zhàn)報(bào)】Bioss抗體新增高分文獻(xiàn)精彩呈現(xiàn)

更新時(shí)間:2024-05-28  |  點(diǎn)擊率:365

截止目前,引用Bioss產(chǎn)品發(fā)表的文獻(xiàn)共29281篇,總影響因子141751.15分,發(fā)表在Nature, Science, Cell以及Immunity等頂級(jí)期刊的文獻(xiàn)共68篇,合作單位覆蓋了清華、北大、復(fù)旦、華盛頓大學(xué)、麻省理工學(xué)院、東京大學(xué)以及紐約大學(xué)等國(guó)際研究機(jī)構(gòu)上百所。

我們每月收集引用Bioss產(chǎn)品發(fā)表的文獻(xiàn)。若您在當(dāng)月已發(fā)表SCI文章,但未被我公司收集,請(qǐng)致電Bioss,我們將贈(zèng)予現(xiàn)金鼓勵(lì),金額標(biāo)準(zhǔn)請(qǐng)參考“發(fā)文章 領(lǐng)獎(jiǎng)金"活動(dòng)頁(yè)面。

近期收錄2024年2月引用Bioss產(chǎn)品發(fā)表的文獻(xiàn)共363篇(圖一,綠色柱),文章影響因子(IF) 總和高達(dá)2298.3,其中,10分以上文獻(xiàn)45篇(圖二)。

【2024年2月文獻(xiàn)戰(zhàn)報(bào)】Bioss抗體新增高分文獻(xiàn)精彩呈現(xiàn)

圖一


【2024年2月文獻(xiàn)戰(zhàn)報(bào)】Bioss抗體新增高分文獻(xiàn)精彩呈現(xiàn)

圖二



本文主要分享引用Bioss產(chǎn)品發(fā)表文章至NatureImmunityCancer Cell等期刊的5篇 IF>15 的文獻(xiàn)摘要,讓我們一起欣賞吧。




Molecular Cancer [IF=37.3]

【2024年2月文獻(xiàn)戰(zhàn)報(bào)】Bioss抗體新增高分文獻(xiàn)精彩呈現(xiàn)

文獻(xiàn)引用產(chǎn)品:bsm-33070M

Ki-67 Mouse mA | IHC

作者單位:重慶醫(yī)科大學(xué)

摘要:CircPDHK1 was upregulated in ccRCC tissues and closely related to WHO/ISUP stage, T stage, distant metastasis, VHL mutation and Ki-67 levels. CircPDHK1 had a functional internal ribosome entry site (IRES) and encoded a novel peptide PDHK1-241aa. Functionally, we confirmed that PDHK1-241aa and not the circPDHK1 promoted the proliferation, migration and invasion of ccRCC. Mechanistically, circPDHK1 was activated by HIF-2A at the transcriptional level. PDHK1-241aa was upregulated and interacted with PPP1CA, causing the relocation of PPP1CA to the nucleus. This thereby inhibited AKT dephosphorylation and activated the AKT-mTOR signaling pathway.



Cellular & Molecular Immunology [IF=24.1]

【2024年2月文獻(xiàn)戰(zhàn)報(bào)】Bioss抗體新增高分文獻(xiàn)精彩呈現(xiàn)

文獻(xiàn)引用抗體:

bs-2789R; Tap1 Rabbit pAb | FC

bs-2374R; TAP2 Rabbit pAb | FC

作者單位:北京大學(xué)

要:CD4+ T cells can "help" or "license" conventional type 1 dendritic cells (cDC1s) to induce CD8+ cytotoxic T lymphocyte (CTL) anticancer responses, as proven in mouse models. We recently identified cDC1s with a transcriptomic imprint of CD4+ T-cell help, specifically in T-cell-infiltrated human cancers, and these cells were associated with a good prognosis and response to PD-1-targeting immunotherapy. Here, we delineate the mechanism of cDC1 licensing by CD4+ T cells in humans. Activated CD4+ T cells produce IFNβ via the STING pathway, which promotes MHC-I antigen (cross-)presentation by cDC1s and thereby improves their ability to induce CTL anticancer responses. In cooperation with CD40 ligand (L), IFNβ also optimizes the costimulatory and other functions of cDC1s required for CTL response induction. IFN-I-producing CD4+ T cells are present in diverse T-cell-infiltrated cancers and likely deliver “help" signals to CTLs locally, according to their transcriptomic profile and colocalization with “helped/licensed" cDCs and tumor-reactive CD8+ T cells. In agreement with this scenario, the presence of IFN-I-producing CD4+ T cells in the TME is associated with overall survival and the response to PD-1 checkpoint blockade in cancer patients.



ADVANCED FUNCTIONAL MATERIALS [IF=19.0]

【2024年2月文獻(xiàn)戰(zhàn)報(bào)】Bioss抗體新增高分文獻(xiàn)精彩呈現(xiàn)

文獻(xiàn)引用產(chǎn)品:bs-4917R

Osteocalcin Rabbit pAb | IF

作者單位:北京大學(xué)口腔醫(yī)院

摘要:Utilization of electro-responsive biomaterials with antibacterial properties is advantageous for facilitating septic wound healing and tissue regeneration. However, the dose-response effects of electrical stimuli from these materials against bacteria are not rigorously characterized, and achieving synergy of bactericidal and pro-regenerative effects of biomaterials remains a major challenge. Here, a graded series of flexible BaTiO3/P(VDF-TrFE) electroactive nanocomposite membranes (EMs) are developed with varying surface charge intensities, to serve as antibacterial dressing for septic wound healing. EMs display broad-spectrum antibacterial effects against both Gram-positive and Gram-negative bacteria in a dose-dependent manner, depending on the magnitude of their surface electrical potential. Mechanistically, the surface charge of EMs increase intracellular levels of reactive oxygen species within bacteria cells, which in turn caused oxidative damage to the bacterial membrane, thereby suppressing bacterial activity and biofilm formation. Moreover, in vivo studies demonstrated that EMs effectively inhibited S. aureus infection and accelerated wound healing in a mouse skin defect model, as well as ameliorated P. gingivalis-mediated periodontal inflammation in a mouse periodontitis model. Hence, this study optimizes the antibacterial properties of electroactive materials and characterizes the dose-response effects of surface electrical charge against bacteria, thus validating the therapeutic applications of electroactive biomaterials in combating bacterial infection.


ACS Nano [IF=17.1]

【2024年2月文獻(xiàn)戰(zhàn)報(bào)】Bioss抗體新增高分文獻(xiàn)精彩呈現(xiàn)

文獻(xiàn)引用抗體:

bs-0292P; BSA-V

D-9106; DAPI

bsm-33070M; Ki-67 Mouse mAb | IF

作者單位:北京大學(xué)

摘要:Cancer progression and treatment-associated cellular stress impairs therapeutic outcome by inducing resistance. Endoplasmic reticulum (ER) stress is responsible for core events. Aberrant activation of stress sensors and their downstream components to disrupt homeostasis have emerged as vital regulators of tumor progression as well as response to cancer therapy. Here, an orchestrated nanophotoinducer (ERsNP) results in specific tumor ER-homing, induces hyperthermia and mounting oxidative stress associated reactive oxygen species (ROS), and provokes intense and lethal ER stress upon near-infrared laser irradiation. The strengthened “dying" of ER stress and ROS subsequently induce apoptosis for both primary and abscopal B16F10 and GL261 tumors, and promote damage-associated molecular patterns to evoke stress-dependent immunogenic cell death effects and release “self-antigens". Thus, there is a cascade to activate maturation of dendritic cells, reprogram myeloid-derived suppressor cells to manipulate immunosuppression, and recruit cytotoxic T lymphocytes and effective antitumor response. The long-term protection against tumor recurrence is realized through cascaded combinatorial preoperative and postoperative photoimmunotherapy including the chemokine (C–C motif) receptor 2 antagonist, ERsNP upon laser irradiation, and an immune checkpoint inhibitor. The results highlight great promise of the orchestrated nanophotoinducer to exert potent immunogenic cell stress and death by reinforcing ER stress and oxidative stress to boost cancer photoimmunotherapy.


Advanced Science [IF=15.1]

【2024年2月文獻(xiàn)戰(zhàn)報(bào)】Bioss抗體新增高分文獻(xiàn)精彩呈現(xiàn)

文獻(xiàn)引用抗體:

bs-0292P-FITC; BSA / FITC | IF

bsm-60235R; Keratin 6 Recombinant Rabbit mAb | IF

作者單位:南方醫(yī)科大學(xué)

摘要:To address current challenges in effectively treating large skin defects caused by trauma in clinical medicine, the fabrication, and evaluation of a novel radially aligned nanofiber scaffold (RAS) with dual growth factor gradients is presented. These aligned nanofibers and the scaffold's spatial design provide many all-around “highways" for cell migration from the edge of the wound to the center area. Besides, the chemotaxis induced by two growth factor gradients further promotes cell migration. Incorporating epidermal growth factor (EGF) aids in the proliferation and differentiation of basal layer cells in the epidermis, augmenting the scaffold's ability to promote epidermal regeneration. Concurrently, the scaffold-bound vascular endothelial growth factor (VEGF) recruits vascular endothelial cells at the wound's center, resulting in angiogenesis and improving blood supply and nutrient delivery, which is critical for granulation tissue regeneration. The RAS+EGF+VEGF group demonstrates superior performance in wound immune regulation, wound closure, hair follicle regeneration, and ECM deposition and remodeling compared to other groups. This study highlights the promising potential of hierarchically assembled nanofiber scaffolds with dual growth factor gradients for wound repair and tissue regeneration applications.





99综合网| 五月婷婷激情网| 日韩激情啪啪| 欧美人妻久久精品二区三区 | 玖玖在线视频| www.久久| 日本操逼视频导航| 日韩欧美综合激情| 天天操夜夜操| 牛牛AV人人夜夜澡人人爽| 亚洲成人综合在线| 777AV电影| 一区二区三区免费岛国片| 人人妻人人狠人人| 麻豆啪啪啪视频| 91婷婷| 青春草莓视频在线观看网址| 亚欧高清v| 少妇一区二区三区高速| 欧亚乱色熟女一区二区| 波多野结衣AV无码一区| 国产精品探花视频| 岛国网址国产 | 丁香五月综合| 色色五月丁香| 成人激情无码在线视频| 夜夜影视四色| 欧美日韩色综合网| 亚洲国产无码精品首页久久久| 97色色网| 九九这里只有精品| 欧美很很操视频| 天天躁日日躁AAAAXXXX国产| wwwcaobibi| 欧美成人性爱视频免费观看| 亚洲国产奇米影视久久| 中文日本免费高清| 少妇无码av专区线| 亚洲伊人a线观看视频| 最新国内自拍av免费| 日韩性爱高清免费视频| 蜜桃av综合网发布| 99re久久| 色婷婷在线视频| 人妻色偷色噜| 黑人精品XXX一区一二区| 综合久久久久久久久91| 97香蕉碰碰人妻国产欧美| 午夜福利视频在线一区| 综合久久中文字幕综合日韩精品| 思思热影视| 高清国产av无码| 六月婷婷综合| 欧美呦呦性爱| 日本在线观看网址| 国产无码三级视频在线观看| 伊人丁香五月婷婷| 无码人妻丰满热妇又大又粗| 日韩性爱电影一区| www.久久久久| 久久久久久久免费A片国产成a人亚洲精∨品无码| 国产小黄片在线免费观看 | 操国产逼| 操逼逼中文字幕| 丰满的三级少妇欧美久久久| 日韩一级欧美一级国产一级台湾| 国产成人拍国产亚洲精品| 逼逼逼逼操操操操操操操操操午夜剧场| 亚洲黄色a级片| 可以看的av| 国产毛片久久久久久久| 日本中文字幕在线视频| 欧美18老人禁| 黄色一级视| 欧美性爱第一页久久| 精品久久久久久中文| 大香蕉520| 一类av片在线看| 女人爽到高潮潮喷18禁网站| 亚洲欧洲日本精品中文a∨| 美女国产一区二区久久| 一级性爱视频免费在线| 极品美女福利在线观看| 日韩免费簧片| www.色操逼| 国产多人在线观看视频| 亚洲图片偷拍视频区| 成人网站 免费观看| 丁香五月av| 中国乱伦一区二区| 国产精品白丝在线播放| 国产亚州日韩欧美看片| 这里只有精品视频在线观看麻豆| 色欲久久综合| 日韩欧美午夜视频在线| 亚洲精品成人动漫在线| 99性爱视频| 国产视频一区二区免费| 日韩性爱1级片视频| 天天干人人看综合| 国产精品秘 福利姬在线观看| 欧美日韩电影成人在线| 人人操,人人液| 99热99色| 亚洲欧美中文日韩视频中国语| 国产成人精品无码久久| 夜夜操美女| 国产操逼网站亚洲一级黄色| 国产精品三级视频网站 | 九九无码视频| 亚洲最大AV网| 熟女人妇一区二区三区| 乱伦图一区| 一级片在线观看高清无码| 欧美真人抽搐一进一出gif | 乱性AV| 亚洲伊人成综合成人网| 激情AV| 婷婷丁香六月天| 大香蕉AV在线| 亚洲色五月| 婷婷五月天激情小说| 亚洲操人| 插入逼91| 黄色人人| 美女极品一区二区三区| 亚洲欧洲精品视频发布| 久久久久久国产手机AV| 国产精品香蕉热久久新品| 成人三级片无码| 九九aV| 热99这里有精品综合久久| 手机在线播放国产福利| 国产人妖的免费的视频| www.av在线视频| Blackedraw视频一区二区| 亚洲欧美一区二区三区一猛片| 色久桃花影院在线观看| 96久久精品一二三区色欲| 色色色色综合网| 天天干天天操天天操夜夜操天天操| 女性喷水高潮在线观看| 色翁荡息又大又硬又粗又爽| 欧美强奸乱能| 成人av免费观看| 乱伦一二三区| 亚洲中文国际强奸字幕| www.99在线| 婷婷在线视频在线观看| 日本一区二区不卡精品| 婷婷五月天av| 国产成人无码a| 国产呦精品一区二区三区下载| 啪啪综合网| 人人操人人干网页| 婷婷五月天AV| 日韩在线一区高清在线| 日本在线激情一区二区三区| 国产黄色视频久久| 人人干人人操人人..com| 性色AV网站| AV免费在线播放一区| 五月丁香久久| 福利伊人玖玖国产| 亚洲伊人久久综合97| 国产亚洲精品第一最新| 美日韩男女操屄视频| 3p国产色噜噜一区| 少妇一区二区三区高速| 久热9| 日本午夜操逼| 三级AV入口| 日本操逼视频免费| 国产一级αv免费看片| 人人看人人插| 99热婷婷| 国产成人bd在线观看| 摸奶性爱视频网站在线免费播放| 高清在线偷拍自拍视频| 99久久婷婷丁香| 亚洲国产美女久久久久| 婷婷91| 国产亚州精品美女久久久免费| 久久久久久久久久久免费精品| 大香网伊人久久综合| 屁屁影院一区二区三区国产| 色黄污美女啪啪啪免费网站| 国产强奸乱伦第1页| 日本道久久综合色色| 亚洲乱码国产乱码精网站| 成年人三级黄色片视频| 久久久久久网址| 九九黄色网| 亚洲中文字幕av| 国产欧美亚洲精品a第2页| 95自拍视频在线观看| 18禁在线视频| 国产精品一区二区久久精品| 开心五月婷婷| 99热久| 97人人草| 激情综合五月| 国产精品精品系列在线观看| 五月婷婷性爱| 丁香五月激情综合| 色色99| 国产h小视频在线观看免费| 五月色综合| 翔田千里AV无码秘 三区| 色婷婷综合网| 亚洲欧美国产va在线播放频| 国产精品999aaa| 国产乱伦视频污| 欧美 精品国产制服第一页| 国产精品国产亚洲区艳妇糸列| 日本成a人v网站在线观看| 婷婷久久五月综合激情| 亚洲人成网站7777| 无码区蜜乳| 999亚洲国产视频| 欧美日本国产日韩激情视频| 91精品无码人妻系列| 十八禁视频网站| 国产91精品在线免费| 狠狠操狠狠操操| 大屁股人妻女教师撅着屁股| 精品国产一区二区三区在线播出| 精品国产一区二区三区香蕉欧美| 岛国网址国产 | 久久精品美女一区| 最新亚洲黄色免费电影 | 9色国产精品一区粉嫩| 欧美在线永久天堂| 天天看天天在线精品| 日本一级二级三级网站| 五月天婷婷成人网| 草草影院日本第一页| 亚洲欧美精品福利在线| 尤物av网站免费在线播放| 岛国视频一二三区| 狠狠色丁香| 99re公开精品免费视频| 超碰九色| 777AV电影| 激情五月激情综合网| 日韩精品在线观看观看| 国产成人无码久久精品| 亚洲天堂五月天国产| 91狼人| 国产黄色影片在线观看| 亚洲国产成人高清在线| 成人乱人伦一区二区| www.99热| 国内毛片国产专区二| 日韩免费av片高清无码| 国产成人自拍视频在线| 激情 欧美 亚洲 小说| 超碰人人草| 亚洲一卡2卡3卡4卡乱码网站 | 午夜成人爽爽爽爽A片李冰冰| 成人aⅴ一区二区三区| 亚洲av噜噜噜噜噜噜| 国产亚州高清国产拍精| 国产不良强奸视频免费看| 婷婷丁香五月激情啪啪| 中国zzijzzijzzwww精品| a片在线播放| 五月天综合网| 欧美日韩操逼嗦吊| 人人人人插| 小日子操bb在线看| 久久久久久99AV无码免费网站| 婷婷久月| 熟女六十路| 国人欧美精品一区二区| 国产精品嫩草久久久久| 青青青国产手线观看视频2| 99色热| 色情五月婷婷| 成人丁香五月| 国产女人成人精品视频| 无码人妻丰满热妇又大又粗| 色黄色美女大长腿午夜视频| 日韩激情无码影院| 亚洲熟妇一,二,三期| 五月婷婷丁香六月| 色色热| 大学生美女口爆| 婷婷五月天基地| 国产v亚洲v日韩v欧美v片另类| 成人电影一区| 99视频内射三四| 五月香婷婷| 成人小说另类在线| 另类图片欧美激情综合| 边做饭边操逼逼| 久久透逼视频| 最新av网站在线观看| 91深夜夜| 国产91精品福利在线| 在线可观看的黄色网址| 欧亚性爱视频免费看| 国产亚洲国产超碰| 狠狠操狠狠| www.色吧5.com| 欧美综合娱乐久久| 免费伦费视频在线观看| 手机看av网站在线看| 亚洲免费看片| 成年人黄色视频免费| 麻豆国产视频精品观看| 伊人久久综合影院| 欧美三级中文字幕hd| 91|九色|国产熟女| 天天日天天操天天射河南省| 国产av激情无码久久天堂| 岛国大片在线观看网站入口| 欧美激情中文字幕另类小说| 亚洲精品三| 亚洲欧洲中文日韩女优乱码| 波多野结衣先锋影音| 久久香蕉影院| 成全在线观看免费观看| 亚洲精品a人片在线观看视| 色情婷婷| 影音先锋视频在线| 色99视频| 国产在线激情视频|